Vitamin D Optimization
It's a hormone that controls thousands of genes. Supplements alone often fail.
Low vitamin D often isn't about sun exposure.
Vitamin D requires multiple activation steps. Each step depends on the same systems that support glutathione, bile flow, and methylation.
When glutathione is depleted
- Oxidative stress damages vitamin D enzymes
- Liver function suffers, reducing bile production
- Vitamin D gets trapped in fat tissue—present but unavailable
VDR is both a vitamin D receptor AND a bile acid sensor.
This creates a sophisticated regulatory network. When vitamin D levels fall, bile acid synthesis becomes dysregulated, reducing bile production. Which further impairs vitamin D absorption.
The vicious cycle: Vitamin D deficiency → reduced bile → worse vitamin D absorption → deeper deficiency
Supplements can't replicate sun exposure.
UVB-synthesized vitamin D provides 90%+ of requirements
Cutaneous synthesis has built-in safety mechanisms—excess converts to inert photoproducts.
Clinical trials show supplements don't match sun benefits
Suggesting critical non-vitamin D pathways from sun exposure.
50,000 IU fails in malabsorption
The same dose works in those with normal digestion. It's not the supplement—it's the system.
Restore the system, not just the number.
Address bile flow, glutathione, and overall metabolic health. Then vitamin D takes care of itself.
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